Search for dissertations about: "Secondary Brain Injury"
Showing result 1 - 5 of 59 swedish dissertations containing the words Secondary Brain Injury.
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1. Intracranial Pressure in Traumatic Brain Injury
Abstract : Traumatic Brain Injury (TBI) continues to be a major problem worldwide. Today, intensive care of patients with TBI is mainely focused on preventing and treating secondary brain injuries. High pressure inside the intracranial cavity (ICP) has been found to be an important feature of disturbed cerebral dynamics and secondary injuries. READ MORE
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2. Brain Tissue Oxygenation in Traumatic Brain Injury : Experimental and Clinical Studies
Abstract : Traumatic brain injury (TBI) is a major cause of death and disability. TBI is frequently followed by cerebral ischemia which is a great contributor to secondary brain damage. The main causes of cerebral ischemia are pathophysiological changes in cerebral blood flow and metabolism. READ MORE
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3. Traumatic brain injury in elderly patients
Abstract : The increase of elderly traumatic brain injury (TBI) patients constitutes a considerable challenge. The aim was therefore to specifically study elderly TBI patients with respect to patient characteristics, neurointensive care (NIC) and outcome, and to identify age specific features, which may be important for selection of patients and optimization of NIC in the elderly. READ MORE
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4. Delayed Cell Death after Traumatic Brain Injury : Role of Reactive Oxygen Species
Abstract : Traumatic brain injury (TBI) is a leading cause of death and disability TBI survivors often suffer from severe disturbances of cognition, memory and emotions. Improving the treatment is of great importance, but as of yet no specific neuroprotective treatment has been found. READ MORE
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5. Oligodendrocyte pathology following Traumatic Brain Injury : Experimental and clinical studies
Abstract : Traumatic brain injury (TBI) caused by traffic and fall accidents, sports-related injuries and violence commonly results in life-changing disabilities. Cognitive impairments following TBI may be due to disruption of axons, stretched by the acceleration/deceleration forces of the initial impact, and their surrounding myelin in neuronal networks. READ MORE