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Showing result 1 - 5 of 130 swedish dissertations matching the above criteria.
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1. Young adults with childhood-onset inflammatory bowel disease - aspects of bone mineral density, body composition and physical exercise
Abstract : Background: Our research group has previously shown that low bone mineral density (BMD) is common in children and adolescents with inflammatory bowel disease (IBD). However, there is limited knowledge on the development of BMD and body composition traits (skeletal muscle and body fat) in early adulthood in this patient group. READ MORE
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2. Molecular biosignatures and gut-barrier alterations in inflammatory bowel diseases
Abstract : Inflammatory bowel diseases (IBD) are chronic immune-mediated disorders affecting the gastrointestinal tract. The multifactorial pathophysiology of IBD is commonly explained as an abnormal interplay between genetic, immune, environmental and microbial factors. READ MORE
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3. Commensal microbes, immune reactivity and childhood inflammatory bowel disease
Abstract : Inflammatory bowel disease (IBD) is characterized by chronic and relapsing intestinal inflammation of unknown etiology, but immune activation by the commensal microbiota probably plays a major role. The two major categories of IBD are ulcerative colitis and Crohn’s disease. READ MORE
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4. Studies of ulcerative colitis with concomitant primary sclerosing cholangitis : Beyond the clinical phenotype
Abstract : Inflammatory bowel disease (IBD) is a group of chronically relapsing immune-related disorders, engaging the gastrointestinal tract. Symptoms vary depending on inflammatory phenotype, but may include diarrhoea, bowel pain and weight loss. The two most common entities are Crohn's disease and ulcerative colitis (UC). READ MORE
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5. Interplay between Pathogenic and Immune Regulatory Mechanisms in G-alpha-i2 deficient colitis
Abstract : The two major forms of inflammatory bowel disease (IBD), Crohn's disease (CD) and ulcerative colitis (UC) are gastrointestinal disorders characterized by chronic and relapsing inflammation. Mice deficient in the inhibitory G protein subunit Gαi2 spontaneously develop chronic colitis and have been used as a model for IBD, with particular similarities to UC. READ MORE