Search for dissertations about: "IFN- g"
Showing result 6 - 10 of 113 swedish dissertations containing the words IFN- g.
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6. Cytokines in Staphylococcus aureus arthritis. For better for worse
Abstract : Septic arthritis is an inflammatory condition in joints caused by in situ residing bacteria accompanied by a septic reaction. Bacteria reach their target via the blood stream and the major human pathogen is Staphylococcus aureus. READ MORE
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7. Immunological mechanisms involved in seasonal pollen allergy and allergen-specific immunotherapy
Abstract : Th1/Th2-like T-cell responses were studied by stimulating peripheral blood mononuclear cells (PBMCs) with allergens or antigens followed by restimulation with mitogens to induce high cytokine synthesis. Allergen-stimulation in vitro induced Th2-skewed cytokine synthesis by PBMCs from birch-pollen-allergic patients. READ MORE
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8. Tumor-targeted superantigens for experimental immunotherapy of human leukemia/lymphoma
Abstract : Bacterial superantigens (SAgs) have the property of stimulating high proportions of T cells expressing certain TCR Vβ sequences. The SAg first binds with high affinity to HLA class II molecules on the target cell and then stimulates the T cell to produce cytokines and to become cytotoxic. READ MORE
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9. Functions of glycoprotein G of herpes simplex virus type 2
Abstract : Background: Herpes simplex virus type 2 (HSV-2) is a common sexually transmitted infection with more than 500 million individuals infected world-wide. A major global health problem is that HSV-2 infection facilitates the spread of HIV. This epidemiological situation warrants the development of a vaccine to reduce the spread of HSV-2. READ MORE
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10. Interplay between Pathogenic and Immune Regulatory Mechanisms in G-alpha-i2 deficient colitis
Abstract : The two major forms of inflammatory bowel disease (IBD), Crohn's disease (CD) and ulcerative colitis (UC) are gastrointestinal disorders characterized by chronic and relapsing inflammation. Mice deficient in the inhibitory G protein subunit Gαi2 spontaneously develop chronic colitis and have been used as a model for IBD, with particular similarities to UC. READ MORE