Search for dissertations about: "Inflammation Mediators"
Showing result 21 - 25 of 318 swedish dissertations containing the words Inflammation Mediators.
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21. Influence of leukotriene D4 and cyclooxygenase-2 on cell survival
Abstract : The pro-inflammatory effect of leukotrienes is an important element in the pathogenesis of multiple disorders, including asthma and inflammatory bowel diseases (IBD). A serious concern for IBD patients is the increased risk of developing colorectal cancer and since there seems to be a general connection between inflammation and cancer we hypothesised that LTD4 induced signalling events could be involved in increasing the tumourgenic potential of cells. READ MORE
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22. Circulating biomarkers in patients with abdominal aortic aneurysm
Abstract : Abdominal aortic aneurysm (AAA) develops in 3-6% of the population over 65 years and affects mainly men. AAA has a complex etiology involving inflammation, proteolysis, fibrinolysis and coagulation. READ MORE
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23. Regulation of survival and proliferation of human intestinal epithelial cells by leukotriene D4
Abstract : The pro-inflammatory mediator, Leukotriene D4 (LTD4) is a product of arachidonic acid cascade and has been implicated in asthma and inflammatory bowel diseases. Prolonged inflammatory conditions like ulcerative colitis increases the risk for the development of cancer and the factors that induce the chronic immune response remain uncertain. READ MORE
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24. Molecular mechanisms in vascular inflammation
Abstract : Vascular inflammation plays an important role in a multitude of major diseases including sepsis and atherosclerosis. Sepsis has been estimated to account for more than 100 000 deaths per year in Europe. READ MORE
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25. Infammatory response to hyperbaric trauma. An experimental and clinical study
Abstract : Reductions in ambient pressure liberates gas in various tissues and can result in decompression illness. In this thesis the patophysiological implications of a blood gas interface resulting from decompression was investigated with reference to the influence on biochemical mediators of inflammation. READ MORE