Search for dissertations about: "Liver carcinogenesis"
Showing result 1 - 5 of 25 swedish dissertations containing the words Liver carcinogenesis.
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1. Chemical carcinogenesis by dimethylnitrosamine : the influence of nutritional status of age
Abstract : Dimethylnitrosamine (DMN) is a potent carcinogen in a variety of animal species. Nitrosamines are widely spread in the environment and many populations of the world are continuously exposed to low doses of them. Dietary deficiency might strongly influence the capacity of the body to metabolize and excrete chemical carcinogens. READ MORE
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2. Hormonal regulation of sex differentiated liver carcinogenesis in the rat
Abstract : Endocrine factors influence cancer development in a variety of reproductive and non-reproductive tissues and organs. The incidence of liver cancer is higher in men than in women and long-term administration of androgens and estrogens has been reported to increase the risk for liver cancer formation, supporting the importance of hormonal factors in the etiology of this tumor. READ MORE
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3. In vivo studies of cell cycle regulating proteins in rats during liver regeneration and during promotion of liver carcinogenesis
Abstract : There are more than one hundred different types of cancer in humans. However, the major platform for all types of cancer is automous and uncontrolled cell division and though most types of cancer are possible to treat and cure by surgery, chemical therapy or irradiation, more efficient and less toxic therapies are urgently needed. READ MORE
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4. Bacteria and cancer : from toxin delivery to carcinogenesis
Abstract : Epidemiological evidence link certain chronic bacterial infections to a higher risk of cancer development. Induction of an inflammatory circuit and the accumulation of genomic instability are considered mechanisms by which bacteria contribute to malignant transformation. READ MORE
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5. Prevention of liver cancer in chronic liver disease : an experimental study of sodium selenite and rat hepatocarcinogenesis
Abstract : Selenium treatment in supranutritional but subtoxic doses has previously been shown to inhibit cell proliferation in preneoplastic lesions in a rat liver carcinogenesis model. The mechanisms are not known, but thioredoxin reductase (TrxR1), a seleno-enzyme essential for maintaining intracellular redox status and cellular defence against oxidative stress might be involved. READ MORE
