Search for dissertations about: "Meningococcal meningitis"
Showing result 6 - 10 of 10 swedish dissertations containing the words Meningococcal meningitis.
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6. Virulence Factors and Motility Mechanisms of Pathogenic Neisseria
Abstract : Neisseria gonorrhoeae and Neisseria meningitidis are two closely related human specific pathogens. Neisseria gonorrhoeae is the causative agent for the sexually transmitted disease gonorrhea and often causes asymptomatic infections in women which is a cause of infertility. READ MORE
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7. Hfq- and sRNA-mediated regulation in Neisseria meningitidis
Abstract : Neisseria meningitidis, also known as the meningococcus, is a human-specific pathogen that commonly colonises the nasopharynx without causing disease. For reasons unknown, N. meningitidis can traverse the nasopharyngeal epithelium and enter the bloodstream, causing invasive meningococcal disease manifesting in septicaemia and/or meningitis. READ MORE
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8. Host responses and bacterial virulence factors in Neisseria infections
Abstract : Neisseria meningitidis and Neisseria gonorrhoeae are human specific pathogens. N. meningitidis is a major cause of meningitis and sepsis, whilst N. gonorrhoeae causes many millions of cases of gonorrhea in the world every year. READ MORE
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9. Receptor Interactions Between Pathogenic Bacteria and Host Cells
Abstract : This thesis focuses on host and pathogen specific interactions during invasive disease. We have investigated the role and impact of different virulence factors of Neisseria gonorrhoeae, N. meningitidis and Streptococcus pyogenes on host epithelial cells and in vivo. N. READ MORE
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10. RNA-mediated gene regulation in Neisseria meningitidis and other nasopharyngeal pathogens
Abstract : Neisseria meningitidis is a transient commensal bacterium of the upper respiratory tract and spreads through respiratory secretions produced by the carrier. While mostly harmless, the bacterium can cause invasive meningococcal disease by entering the circulatory system, leading to septicaemia and/or meningitis. READ MORE