Search for dissertations about: "allergic sensitisation"
Showing result 1 - 5 of 31 swedish dissertations containing the words allergic sensitisation.
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1. Allergic Disease among Adolescents, IgE-sensitisation, Health-Related Quality of Life and physical activity
Abstract : .... READ MORE
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2. Epoxides as Contact Allergens - Formation, Sensitising Potency and Structure-Activity Relationships
Abstract : Chemicals in our environment with which we have repeated skin contact can cause skin sensitisation (contact allergy). To trigger an immune response a compound (hapten) must be able to penetrate the skin, where it needs to be reactive enough to bind to proteins and form immunogenic complexes that are recognised as foreign. READ MORE
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3. Early life cytokines, viral infections and IgE-mediated allergic disease
Abstract : Background: The reasons why some individuals become IgE-sensitised and allergic are largely unknown, though genetic- and early life environmental factors seem to be of importance.Objective: The overall aim of this thesis was to investigate the relationship between IgE-sensitisation and allergic disease, viral infections, genetic markers and early life cytokines. READ MORE
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4. Development of allergy, salivary IgA antibodies and gut microbiota in a Swedish birth cohort
Abstract : The increasing prevalence of allergic diseases in affluent societies has been associated with changes in microbial exposure early in life and a less diverse gut flora. The objective of this thesis was to assess the development of allergic sensitisation and symptoms during the first four years of life in a non-selected birth cohort in relation to environmental factors, family history, gut microbiota and salivary IgA antibodies. READ MORE
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5. Epidemiology of asthma in primary school children
Abstract : Background: Childhood asthma has increased worldwide, although recent studies report a prevalence plateau in some western countries.Aims: To investigate the prevalence of asthma and the associated risk factor patterns from ages 7-8 to 11-12 with special emphasis on the hereditary component, and further to study prevalence trends at age 7-8 from 1996 to 2006 and the possible determinants of these trends. READ MORE