Search for dissertations about: "c reactive protein in trauma"
Showing result 1 - 5 of 6 swedish dissertations containing the words c reactive protein in trauma.
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1. Experimental Traumatic Brain Injury and Cell Death - in vivo and in vitro aspects
Abstract : Traumatic and ischemic brain damage are major causes of disability and death. While much effort has been spent on developing pharmacological treatments for these conditions, no neuroprotective drugs are in clinical use. READ MORE
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2. Mechanisms of traumatic brain injury in the rat : Morphological consequences and neurotrophic responses
Abstract : Traumatic brain injury is a major cause of death and disability in the Western world. especially in young people. Understanding the pathophysiology of traumatic brain injury is the basis for further development of better therapeutic strategies for such patients. READ MORE
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3. Healthcare associated infections in vascular surgery
Abstract : Health care associated infections (HCAI) affect roughly four million people in Europe annually, resulting in 16 million extra days of hospital stay, 37 000 attributable deaths and direct costs of approximately € 7 billion. HCAI in vascular surgery are relatively common complications with potentially devastating consequences. READ MORE
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4. The role of glucocorticoid receptors and mitogen-regulated protein kinases in the cochlea
Abstract : Hearing loss induced by acoustic trauma is a common handicap for the human population. Acoustic trauma triggers a cascade of changes in the cochlea such as a direct mechanical damage of sensory hair cells, biochemical disturbances including reactive oxygen and nitrogen species generation, release of pro-inflammatory cytokines and exitotoxicity. READ MORE
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5. Assessment of experimental neurotrauma
Abstract : Model systems of closed head injury are designed to replicate aspects of traumatic brain injury, ischemia and epilepsy in man. The aim of this thesis was to (1) develop sensitive systems enabling analyses of the biochemical reactions, as reflected by leakage of marker compounds to the cerebrospinal fluid (CSF); and (2) develop and assess a new model for closed head injury and associated dynamic changes in the brain cell. READ MORE