Search for dissertations about: "opportunistic viral infection"
Showing result 1 - 5 of 13 swedish dissertations containing the words opportunistic viral infection.
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1. Opportunistic viral infection after paediatric transplantation
Abstract : Background: Opportunistic viral infections can cause considerable morbidity and mortality in organ and stem cell transplanted (SCT) patients, mainly due to iatrogenic T cell dysfunction. Whereas in SCT patients, in general the immunosuppressive treatment can be discontinued after 6-12 months, for the majority of organ transplanted patients, the need for treatment is life-long. READ MORE
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2. Innate immunity in the immunopathogenesis of chronic viral infection
Abstract : Natural killer (NK) cells have a key role in control and clearance of viral infections. To carry out this function NK cells are capable of recognising infected cells and responding with induction of apoptosis in these cells, and production of pro-inflammatory cytokines. READ MORE
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3. Molecular dynamics in HIV-1 infection of the brain
Abstract : The focus of the present thesis was to investigate the molecular dynamics in HIV-l infection of the brain. HIV-l infects the brain early during the infection and causes neurological syndromes and neuropathological modifications. READ MORE
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4. On HIV-1 infection in the central nervous system
Abstract : HIV-1 (hereafter referred to as HIV) is a neurotropic virus that invades the central nervous system (CNS). The CNS is then susceptible to an acute infection and later to CNS opportunistic diseases and AIDS dementia complex (ADC). HIV does not infect the neurons, and neuropathogenesis is largely mediated by the immune system. READ MORE
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5. Studies of innate immune stimulation with CpG in HIV infection
Abstract : Two types of human immunodeficiency virus (HIV) may cause acquired immune deficiency syndrome (AIDS): HIV-1, the most pathogenic virus of the two and found worldwide, and HIV-2, which is mostly detected in West Africa and known to be less transmissible and which demonstrates slower progression towards immunodeficiency. How CD4+ T cells are lost during these two infections is not yet completely clear; however, a constant battle against the rapid viral turnover, resulting in chronic inflammation, is thought to exhaust several compartments of the immune system. READ MORE