Search for dissertations about: "streptokocker"
Showing result 21 - 25 of 36 swedish dissertations containing the word streptokocker.
-
21. Group A Streptococci; Epidemiology and Genetics of Antibiotic Resistance
Abstract : Group A streptococci (GAS) cause throat and skin infections, severe invasive diseases as well as the non-suppurative complications acute rheumatic fever and glomerulonephritis. GAS have retained susceptibility to betalactam drugs, but resistance to alternative antibiotics is of concern. READ MORE
-
22. Surface proteins of pathogenic streptococci
Abstract : Streptococcus pyogenes (group A streptococcus) and the group B streptococcus (GBS) are two important human pathogens that cause different types of diseases and express different surface structures implicated in virulence. This thesis focuses on several surface proteins expressed by these pathogens, analyzing the biological function of these proteins and their ability to elicit protective immunity. READ MORE
-
23. Interactions of human C4BP with Bordetella pertussis and Streptococcus pyogenes
Abstract : Many microorganisms have developed mechanisms to protect themselves against attack from the complement system of the host. One possible mechanism for a microorganism to evade complement attack is to bind a human complement regulator, which may allow the microorganism to down-regulate complement activation. READ MORE
-
24. Recurrent Streptococcal Pharyngotonsillitis Studies on etiology and treatment
Abstract : In acute pharyngotonsillitis group A streptococci (GAS) is the etiological agent in 30-50% of cases. Phenoxymethylpenicillin (pcV) is the drug of choice in Sweden. However, the failure rate is as high as 5-25% and further pcV treatment is followed by still higher failure rates. The background of failures remains largely elusive. READ MORE
-
25. Interactions between Streptococcus pyogenes and the human immune system, with special reference to C4b-binding protein
Abstract : The ability of many pathogenic microorganisms to cause infection is dependent on their ability to evade attack by the complement system of the host. One mechanism by which microorganisms may avoid complement attack is to bind complement inhibitory molecules present in the host. READ MORE