Search for dissertations about: "copd in smokers"
Showing result 1 - 5 of 45 swedish dissertations containing the words copd in smokers.
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1. Proteolytic imbalance in COPD : epidemiological and clinical aspects
Abstract : Background: The complete pathologic mechanism behind the development of chronic obstructive pulmonary disease (COPD) remains unclear, but several risk factors have been identified, of which smoking is the most common. Proteolytic imbalance contributes to lung tissue degradation and is related to both smoking and COPD symptoms. READ MORE
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2. Prevalence, risk factors and comorbidities of COPD among never-smokers
Abstract : The overall aim of this thesis was to investigate prevalence, risk factors and comorbidity of COPD among never-smokers. COPD was in this thesis defined as a syndrome also including prior asthma diagnosis with chronic airway obstruction. READ MORE
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3. Chronic obstructive pulmonary disease (COPD) : prevalence, incidence, decline in lung function and risk factors
Abstract : The Obstructive Lung Disease in Northern Sweden (OLIN) Studies started in 1985 as an epidemiological project with the aim to detect preventable risk factors for obstructive lung diseases and allergy. In recent years there has been a focus also on obstructive sleep apnoea syndrome (OSAS) and chronic obstructive pulmonary disease (COPD) besides asthma and allergy. READ MORE
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4. Lung function in smokers - Aspects on COPD diagnosis and associations to atherosclerosis and alcohol consumption
Abstract : Smoking is the most common and important risk factor for reduced lung function. Chronic Obstructive Pulmonary Disease (COPD) affects mainly smokers and is characterized by airflow obstruction, assessed using one of two major diagnostic spirometric criteria. READ MORE
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5. New Aspects of Alveolar Adaptive Immune Responses in COPD
Abstract : Chronic obstructive pulmonary disease (COPD) is characterised by a chronic inflammation in conducting airways and the alveolar parenchyma. The inflammation, which is a consequence of inhalation of noxious gases like tobacco smoke, has traditionally been thought to be driven by innate immune responses. READ MORE