Search for dissertations about: "thesis in staining"

Showing result 1 - 5 of 460 swedish dissertations containing the words thesis in staining.

  1. 1. Cytokines and immune balance in preeclampsia a survey of some immunological variables and methods in the study of preeclampsia

    University dissertation from Institutionen för molekylär och klinisk medicin

    Author : Yvonne Jonsson; Christina Ekerfelt; Göran Berg; Leif Matthiesen; Lucia Mincheva-Nilsson; [2005]
    Keywords : MEDICIN OCH HÄLSOVETENSKAP; MEDICAL AND HEALTH SCIENCES; Preeclampsia; Cytokines; Leukocytes; Immunology; Placenta; Th1 Th2 balance; Inflammation; MEDICINE Microbiology; immunology; infectious diseases Immunology Immunobiology; MEDICIN Mikrobiologi; immunologi; infektionssjukdomar Immunologi Immunbiologi;

    Abstract : Preeclampsia is one of the most feared pregnancy complications, with a risk of maternal and fetal death and with no ideal therapy readily available. The cause of this strictly pregnancyrelated disease is still unknown and is therefore a great challenge to all researchers in the field of pregnancy-related pathophysiology. READ MORE

  2. 2. Apoptosis and tumor cell death pathways in response to HAMLET

    University dissertation from Department of Laboratory Medicine, Lund University

    Author : Oskar Hallgren; [2005]
    Keywords : MEDICIN OCH HÄLSOVETENSKAP; MEDICAL AND HEALTH SCIENCES; MEDICIN OCH HÄLSOVETENSKAP; MEDICAL AND HEALTH SCIENCES; Biomedicinska vetenskaper allmänt ; General biomedical sciences; Klinisk biologi; apoptosis; autophagy; alpha-lactalbumin; Clinical biology; programmed cell death; HAMLET; cell cycle;

    Abstract : HAMLET (Human alpha-lactalbumin made lethal to tumor cells) is a molecular complex of alpha-lactalbumin and oleic acid, that induces programmed cells death in tumor cells but leaves healthy differentiated cells unaffected. It has a broad anti-tumor activity as more than 40 cell lines from different origin are sensitive. READ MORE

  3. 3. Mechanisms of HAMLET-induced cancer cell death

    University dissertation from Division of Microbiology, Immunology and Glycobiology - MIG

    Author : Sonja Aits; [2010]
    Keywords : MEDICIN OCH HÄLSOVETENSKAP; MEDICAL AND HEALTH SCIENCES; MEDICIN OCH HÄLSOVETENSKAP; MEDICAL AND HEALTH SCIENCES; alpha-actinin; prohibitin; alpha-lactalbumin; HAMLET; cancer; cell death; autophagy; focal adhesions; c-Myc; hexokinase; glycolysis;

    Abstract : HAMLET, a complex of α-lactalbumin and oleic acid, preferentially kills cancer cells and is also effective in vivo. HAMLET causes apoptosis but cells die even if this pathway is inhibited. Thus, the role of autophagy, an alternative cell death pathway, was examined. READ MORE

  4. 4. MHC class I-induced apoptosis in hematopoietic cells

    University dissertation from Marie Wallén Öhman, Sotarevägen 10, S-227 30 Lund, Sweden

    Author : Marie Wallén Öhman; [1996]
    Keywords : MEDICIN OCH HÄLSOVETENSKAP; MEDICAL AND HEALTH SCIENCES; serology; Immunology; Bioteknik; Biotechnology; antibody; CD40; Myeloid cells; B-cells; HLA class I; cell death; transplantation; Immunologi; serologi;

    Abstract : The aims of this study have been focused on the identification of cell surface structures involved in the induction and regulation of apoptosis. Monoclonal antibodies against the major histocompatibility complex class I (MHC-I) were shown to induce apoptosis when cross-linked on the cell surface of different malignant cells having a pre-B or a myeloid precursor phenotype. READ MORE

  5. 5. Modeling Amyloid Disease in Drosophila melanogaster

    University dissertation from Linköping : Linköping University Electronic Press

    Author : Ina Berg; Per Hammarström; Stefan Thor; Damian Crowther; [2010]
    Keywords : NATURVETENSKAP; NATURAL SCIENCES; NATURAL SCIENCES; NATURVETENSKAP;

    Abstract : Amyloid diseases are caused by protein misfolding and aggregation. To date there are 27 known proteins causing amyloid disorders involving brain and peripheral protein deposition. The proteins involved in this mechanism do not share sequence homology, but the amyloid fibrils share biophysical properties and possibly a common pathogenic mechanism. READ MORE